By Karen Hassey Dow
Collage of principal Florida, Orlando.Text on breast melanoma for nurses and healthiness care execs. Discusses clinical administration, nursing help, and relations matters relating breast melanoma. 26 U.S. participants.
Read Online or Download Contemporary Issues in Breast Cancer (Jones and Bartlett Series in Oncology) PDF
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Extra resources for Contemporary Issues in Breast Cancer (Jones and Bartlett Series in Oncology)
B Cancers that cluster in families. c Genetic syndromes. or mutated in different types of cancer. It is also possible that there may be an ordered or sequential loss of tumor-suppressor genes as a cell progresses from predisposed to overtly cancerous. Finally, several chromosomal regions also harbor oncogenes. , 1994). It is estimated that 15% of all cancer cases are thought to be familial cancers. Familial cancers are defined as cancers that occur in families at greater than expected rates. Since cancer is a common disease, separating familial from nonfamilial cancers is not always simple.
Proto-oncogenes can be altered as a result of a mutation in the gene itself or by an external control. The different mechanisms for proto-oncogene activation are thought to lead to gain-of-function changes that enhance the positive signals for cell growth. Oncogenes have a dominant effect at the cellular level, that is, when activated, a single mutant area on one chromosome is sufficient to change the function of the cell from normal to malignant. Over 50 different proto-oncogenes are now known, and of these, about 20 have actually been found in mutant form in human tumors (Weinberg, 1994).
The result of this type of cancer gene is an intrinsic genetic instability (Cleaver, 1994). DNA repair processes are indispensable for protection from spontaneous and environmentally induced cancers. A Page 10 Table 1-2 Selected Examples of Tumor - Suppressor GenesDisorders in Which Gene AffectedTumor-Suppressor GenesFamilialSporadicRB1RetinoblastomaLung cancerWT1Wilms tumorLung cancerP53Li-Fraumeni syndromeMany epithelial carcinomasNF1Neurofibromatosis, type 1UnknownDCCUnknownColorectal cancer defect in DNA repair is a major contributor to this observed genetic instability.